
Is obesity primarily mediated by deficiencies in GABA, GABA transmission and/or GABA reception?
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In other words, are most cases of obesity caused by GABAergic factors, such as:
insufficient quantities of the neurotransmitter GABA, or
too much reabsorption of GABA, or
GABA receptors not responding to GABA?
I am planning to write a paper on this hypothesis (unless someone disproves it in the comments).
This question is managed and resolved by Manifold.
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